Taipei Medical University Institutional Repository:Item 987654321/6310
English  |  正體中文  |  简体中文  |  全文笔数/总笔数 : 45069/58245 (77%)
造访人次 : 2342376      在线人数 : 219
RC Version 7.0 © Powered By DSPACE, MIT. Enhanced by NTU Library IR team.
搜寻范围 查询小技巧：
  • 您可在西文检索词汇前后加上"双引号"，以获取较精准的检索结果
  • 若欲以作者姓名搜寻，建议至进阶搜寻限定作者字段，可获得较完整数据
    •  进阶搜寻


    jsp.display-item.identifier=請使用永久網址來引用或連結此文件: http://libir.tmu.edu.tw/handle/987654321/6310


    题名: 子宮內膜異位病人腹腔液中抑制胚胎發育因子的分析
    Identification of Embryo-toxic Factor in Peritoneal Fluid from Endometriosis Patients
    作者: 吳振森
    Jenn Sen Wu
    贡献者: 藥學研究所
    关键词: 子宮內膜異位症
    抑制胚胎發育因子
    腹腔液
    Endometriosis
    Embryo-toxic factor
    Peritoneal fluid
    日期: 1993
    上传时间: 2009-09-11 16:50:54 (UTC+8)
    摘要: 子宮內膜異位症在生育年齡婦女是常見的疾病，近來許多研究報告指出，
    內膜異位病人的腹膜滲出液會抑制兩細胞胚胎的繼續發育。本研究的目的
    在於標定出內膜異位症病人的腹腔液中抑制胚胎發育的因子，尋求其做為
    內膜異位病人在診斷及治療的評估上，提供一個指標。實驗結果從電泳分
    析發現子宮內膜異位病人在腹腔液中含有一個分子量110 kDa 的蛋白質，
    同時這些病人的腹腔液有顯著地抑制胚胎發育的作用。其次使用分子過篩
    管柱層析依分子量大小分離腹腔液成三個部份，其中又以分子量 147-54
    kDa 的部份有明顯的胚胎抑制作用，因此在本研究指出該110 kDa蛋白質
    可能是內膜異位症病人的腹腔液中抑制胚胎發育的因子。
    Endometriosis is a common disease in the population of
    reproductive age. Recent reports indicated that peritoneal
    fluid(PF) from patients with endometriosis significantly
    inhibited two-cell embryos development. The purpose of this
    study is to identify the embryo-toxic factor(s) and to assess
    the possibility of testing the factor(s) to predict prognosis
    and treatment effect in infertile women with endometriosis. The
    results indicated that a 110 kDa protein was detected in PF
    from women with endometriosis by gel electrophoresis. The PF
    from these patients significantly inhibited embryo development.
    Three molecular weight(MW) fractions were obtained by gel
    permeation chromatography,and MW fraction 147-54 kDa showed
    apparent inhibition of embryo development. This study suggests
    that the 110 kDa protein seems to be the embryo-toxic factorin
    PF from endometriosis patients.
    数据类型: thesis
    显示于类别:[藥學系] 博碩士論文

    文件中的档案:

    档案 描述 大小格式浏览次数
    摘要.doc26KbMicrosoft Word110检视/开启
    摘要.pdf721KbAdobe PDF107检视/开启
    摘要.ppt116KbMicrosoft Powerpoint88检视/开启
    摘要.ps804KbPostscript76检视/开启


    在TMUIR中所有的数据项都受到原著作权保护.

    TAIR相关文章

    著作權聲明 Copyright Notice

    • 本平台之數位內容為臺北醫學大學所收錄之機構典藏，包含體系內各式學術著作及學術產出。秉持開放取用的精神，提供使用者進行資料檢索、下載與取用，惟仍請適度、合理地於合法範圍內使用本平台之內容，以尊重著作權人之權益。商業上之利用，請先取得著作權人之授權。

      The digital content on this platform is part of the Taipei Medical University Institutional Repository, featuring various academic works and outputs from the institution. It offers free access to academic research and public education for non-commercial use. Please use the content appropriately and within legal boundaries to respect copyright owners' rights. For commercial use, please obtain prior authorization from the copyright owner.

    • 瀏覽或使用本平台，視同使用者已完全接受並瞭解聲明中所有規範、中華民國相關法規、一切國際網路規定及使用慣例，並不得為任何不法目的使用TMUIR。

      By utilising the platform, users are deemed to have fully accepted and understood all the regulations set out in the statement, relevant laws of the Republic of China, all international internet regulations, and usage conventions. Furthermore, users must not use TMUIR for any illegal purposes.

    • 本平台盡力防止侵害著作權人之權益。若發現本平台之數位內容有侵害著作權人權益情事者，煩請權利人通知本平台維護人員([email protected])，將立即採取移除該數位著作等補救措施。

      TMUIR is made to protect the interests of copyright owners. If you believe that any material on the website infringes copyright, please contact our staff([email protected]). We will remove the work from the repository.

    Back to Top
    DSpace Software Copyright © 2002-2004  MIT &  Hewlett-Packard  /   Enhanced by   NTU Library IR team Copyright ©   - 回馈